By R. K. Ross MD, G. A. Coetzee PhD (auth.), Zbigniew Petrovich MD, Luc Baert MD, Luther W. Brady MD (eds.)

The booklet offers accomplished insurance of techniques within the administration of capsula-confined and metastatic endocrine-responsive and endocrine-unresponsive prostate melanoma. in addition to special chapters at the complete diversity of therapies, there are heritage chapters at the epidemiology, improvement, and traditional historical past of the untreated illness. The arguable factor of the worth of prostate-specific antigen as a screening instrument is usually totally mentioned. All chapters were written through the world over well-known specialists of their fields. This quantity is an authoritative evaluate of therapy techniques that might entice all excited by the prognosis and remedy of carcinoma of the prostate.

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In the first year of the study approximately 87% of the men screened had a PSA below 4 ng/ml, and 67% had a PSA below 2 ng/ml. Due to the low PPV of DRE and TRUS many biopsies were taken from men with a PSA below 4 ng/ml. The number of cancers is very limited. For the future it is expected that the protocol will be slightly altered. Those men with a PSA below a certain cutoff (still to be determined) will not be investigated with DRE and TRUS. With this policy, many men will not need to be examined, and more importantly, fewer biopsies will need to be taken.

C Mucinous carcinoma (X 100). D Small cell undifferentiated carcinoma of the prostate is highly malignant, is frequently of advanced stage at presentation, responds poorly to antiandrogen therapy, and has a poor prognosis, with a median survival of less than 2 years (OESTERLING et al. 1992). 5 Transitional Cell Carcinoma Transitional cell carcinoma of the prostate is histologically indistinguishable from bladder cancer. M. Van de Voorde (X 100). E Transitional cell carcinoma with stromal invasion (X 100).

Basal cells may demonstrate immunophenotypic myoepithelial differentiation in related benign proliferative conditions, such as sclerosing adenosis and florid basal cell hyperplasia, in spite of the absence of this feature in the normal prostate (VAN DE VOORDE et al. 1994a). cells occur in all areas of the prostate but tend to be more abundant in the major ducts and are more irregularly distributed throughout acinar tissue (ABRAHAMSSON et al. 1986). 0001) of neuroendocrine cells than the epithelium in the transition zone, with a 6/1000 ratio (unpublished data).

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