By Maria A. Ron, Trevor W. Robbins

Lately there were significant advances in components of scientific neuroscience together with neurogenetics, neuroimaging and the clinical examine of recognition. problems of mind and brain II brings jointly crucial findings because the prior quantity was once released in 1998. Imaging of the conventional and irregular brain figures prominently, and there's assurance of genes and behaviour, mind improvement, recognition and aggression. New disease-oriented chapters aspect contemporary advances in dementia, affective disease and drug use and abuse. medical chapters are paired with these describing neuropathology or experimental types of affliction.

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Additional info for Disorders of Brain and Mind: Volume 2

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A study of developmental speech and language disorders in twins. J Speech Hear Res, 35, 1086–94. McClearn GE, Johansson B, Berg S et al. (1997). Substantial genetic influence on cognitive abilities in twins 80+ years old. Science, 276, 1560–63. McGue M, Bouchard TJ Jr, Iacono WG and Lykken DT (1993). Behavioral genetics of cognitive ability: a life-span perspective. In Nature, Nurture, and Psychology, ed. R Plomin and GE McClearn, pp. 59–76. Washington, DC: American Psychological Association. Neisser U, Boodoo G, Bouchard TJ Jr et al.

1991; Grigorenko et al. 1997; Schulte-K¨orne et al. 1997). The next step is to pin down the specific genes responsible for these QTL regions (Smith et al. 1998). When the specific genes are identified (so far, the QTL linkage has only been tracked to its neighbourhood of several million base pairs of DNA rather than to a specific location), it will be of great interest to investigate the 22 R Plomin extent to which the gene’s effects are specific to reading, or affect language or other cognitive processes more broadly.

Broad heritability can of course be established with the aid of twin studies, which give us useful but often nonspecific information about the nature of the genetic components of complex disorders. For a deeper understanding of the genetic predisposition to psychiatric conditions, it may be more profitable to seek the genetic basis of endophenotypes that are common to a number of diseases or disorders and which reflect the neural substrate underlying symptoms rather than syndromes. Hence, if a clinical disorder is genetically and phenotypically complex, we might ask, could each genetic locus contribute to distinct aspects of the illness phenotype?

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